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Exposure to noise and ototoxic drugs are responsible for much of the debilitating hearing loss experienced by about 350 million people worldwide. Beyond hearing aids and cochlear implants, there have been no other FDA approved drug interventions established in the clinic that would either protect or reverse the effects of hearing loss. Using Auditory Brainstem Responses (ABR) in a guinea pig model, we demonstrate that fluvastatin, an inhibitor of HMG-CoA reductase, the rate-limiting enzyme of the mevalonate pathway, protects against loss of cochlear function initiated by high intensity noise. A novel synchrotron radiation based X-ray tomographic method that imaged soft tissues at micrometer resolution in unsectioned cochleae, allowed an efficient, qualitative evaluation of the three-dimensional internal structure of the intact organ. For quantitative measures, plastic embedded cochleae were sectioned followed by hair cell counting.

Protection in noise-exposed cochleae is associated with retention of inner and outer hair cells. This study demonstrates the potential of HMG-CoA reductase inhibitors, already vetted in human medicine for other purposes, to protect against noise induced hearing loss. Sadhak bamakhyapa serial watch online.

Hearing loss can be caused by exposure to sound levels that damage the inner ear,,,,,,,,,,,,,, iatrogenic intervention such as treatment with ototoxic drugs,,,, or diseases including infections and genetic syndromes,,,,. Free download ghost recon future soldier apk. The impact of hearing loss on our society is reflected by the fact that one in six Americans exhibits a mild-to-severe hearing loss. An associated impact on earning power as well as on societal integration is documented by a variety of review papers. Despite the apparent problem, there are no drugs that are FDA approved to protect, rescue, or repair cochlear damage. Today, treatment of hearing loss is typically limited to amplification of the sound signal (hearing aids) or the surgical implantation of cochlear implants in severe-to-profound deafness. Systemic or local application of drugs has been used with only limited success. Emerging approaches to treatment methods include gene manipulation and stem cell therapy, but none are yet viable for human hearing medicine.

In the cochlea of a normal hearing subject, soft tissue structures transform acoustically induced vibrations into trains of action potentials, which are carried to the brain where they are perceived as sound. Outer and inner hair cells play a key role in this mechano-electrical transduction,,,. Hair cells are the primary receptor cells in the cochlea and are connected via bipolar spiral ganglion neurons to the brain stem. Noise exposure, depending on intensity and duration, can cause damage or loss of the hair cells, of the neuron-hair cell synapses, and/or degeneration of neurons, all of which have the potential to cause hearing impairment. Since neither hair cells nor spiral ganglion neurons and their synapses are capable of regenerating spontaneously, the damage is permanent.